Ulcer - stomach; Peptic disease; Stomach ulcer
For people with Helicobacter pylori infection, the main goal is to get rid of the bacteria that causes the infection. Many different medicines work. They usually include either an H2 receptor antagonist such as famotidine (Pepcid) or nizatidine (Axid) or a proton pump inhibitor such as omeprazole (Prilosec) or esomeprazole (Nexium) to suppress acid, combined with two antibiotics.
After you finish your medicines, your doctor will likely order a test to make sure that the H. pylori infection is gone.
Those who do not have an H. pylori infection may be prescribed ulcer-healing medications such as antacids, H2 receptor antagonists, or proton pump inhibitors. Long-term treatment may be needed.
If the ulcer bleeds, endoscopy can control bleeding in most cases.
Surgery may be recommended for persons who do not respond to medicines or endoscopy. Surgical procedures for gastric ulcers includ...
Generic Name: OMEPRAZOLE/SODIUM BICARBONATE - ORAL Pronounced: (oh-MEP-ra-zole/SOE-dee-um bye-KAR-bo-nate) Omeprazole-sodium Bicarbonate Oral Uses
This medication is a combination of omeprazole and sodium
bicarbonate. It is used to treat certain stomach and esophagus problems (such
as acid reflux, ulcers). It is also used to prevent stomach bleeding in very
Omeprazole works by decreasing the amount of acid your
stomach makes. It belongs to a class of drugs known as proton pump inhibitors
(PPIs). Sodium bicarbonate is an antacid that reduces stomach acid and helps
omeprazole to work better. This medication relieves symptoms such as heartburn,
difficulty swallowing, and persistent cough. It helps heal acid damage to the
stomach and esophagus, helps prevent ulcers, and may help prevent cancer of the
If you are self-treating with this medication,
over-the-counter omeprazole/sodium bicarbonate products are used t...
Causes In 1982, two Australian scientists identified H. pylori as the main cause of stomach ulcers. They showed that inflammation of the stomach and stomach ulcers result from an infection of the stomach caused by H. pylori bacteria. This discovery was so important that the researchers were awarded the Nobel Prize in Medicine in 2005. The bacteria appear to trigger ulcers in the following way: H. pylori's corkscrew shape enables them to penetrate the mucus layer of the stomach or duodenum so that they can attach themselves to the lining. The surfaces of the cells lining the stomach contain a protein, called decay-accelerating factor, which acts as a receptor for the bacteria. H. pylori survive in the highly acidic environment by producing urease, an enzyme that generates ammonia to neutralize the acid. H. pylori stimulate the increased release of gastrin. Higher gastrin levels promote increased acid secretion. The increased acid damages the intestinal lining, leading to ulcers in certain ...
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