Someone posted the exact same long-winded rant about diabetes companies and stem cell research at several other blogs (not this one!). Although the author has some good points, others are purely misinformation. I just can't avoid commenting on one: he/she has the perverse notion that insulin "does not prevent the catastrophic side effects" of diabetes. There is plenty of information that insulin can indeed prevent catastrophic side effects. The most obvious example is DKA: without insulin, diabetic ketoacidosis is the most catastrophic side effect of all, routinely resulting in death. Appropriate instructions to patients with T1DM to never withhold insulin therapy, even when sick and vomiting, has resulted in a dramatic decrease in this complication. The DCCT , published way back in 1993, showed that tight control of blood sugar (with insulin, meal planning, exercise, and frequent BG testing) in people with T1DM resulted in an overwhelming difference in the chan...
Generic Name: INSULIN REGULAR HUMAN - INJECTION Pronounced: (IN-sue-lin) Insulin Regular Human Inj Uses
This man-made insulin product is identical to human
insulin. It is used to treat diabetes mellitus. Like other insulin products, it
works by helping sugar (glucose) get into cells. It is a short-acting
This insulin is usually used in combination with a medium-
or long-acting insulin product injected under the skin to control high blood
sugar. In some people with diabetes, insulin may be used alone or with oral
diabetes drugs (e.g., sulfonylureas like glyburide or
Even with diabetes, you can lead an active and healthy
life if you eat a balanced diet, exercise regularly, and take your insulin as
directed. Controlling high blood sugar helps prevent kidney damage, blindness,
nerve problems, loss of limbs, and sexual function problems. Proper control of
diabetes may also lessen your risk of a heart attack or st...
The American Diabetes Association (ADA) has recently voiced concerns about pancreatic side effects of several diabetes drugs, all of which are related in one way or another to two gut hormones called incretins. The incretins, GLP-1 (glucagon-like peptide-1) and GIP (gastric inhibitory peptide) lower blood glucose levels by increasing insulin secretion from the beta cells of the pancreas.
Drugs that mimic the blood-glucose lowering effects of GLP-1 have been developed, and are called incretin mimetics or GLP-1 receptor agonists. The three that are presently approved by the FDA are Byetta (exenatide), Victoza (liraglutide), and Bydureon (long-acting exenatide).
Both incretin hormones are rapidly inactivated by an enzyme called DPP-4 (dipeptidyl peptidase-4). Drugs have been developed that inhibit DPP-4, and thereby enhance the effect of the incretins to reduce BG levels. At least five DPP-4 inhibitors have been developed, and four are currently available in the US: Ja...
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