Klonopin is an anti-anxiety drug that's often prescribed for anxiety accompanying bipolar disorder, panic disorder and some seizure disorders. Its generic name is clonazepam. Klonopin works by lowering abnormal electrical activity in the brain.
This drug is also sometimes prescribed to treat symptoms of akithisia , a problem with physical restlessness that can occur as a side effect of antipsychotic medications and a few other drugs.
It's important that your doctor and pharmacist know about all prescription and over-the-counter drugs, herbal products (especially St. John's wort ), and all supplements you're taking. There is a long list of medications that might interact badly with Klonopin. The most common are:
Cold and allergy medications
For a more complete list, see Clonazepam under "What special precautions should I follow?"
I asked this question of people who have bipolar disorder and got a wide variety of answers, ranging from, "I pop them like Tic-Tacs so I am only prescribed five at a time," to "It mellows me out," to " It makes me want to crawl out of my skin *shudders*." Here are some of the other answers:
I get very sleepy with it so I take only a half. Every person is different with medicines so your body may react differently then other people's bodies. It's a good medicine to take.
I take them before I go shopping makes it easier for me less anxiety.
The worst side effect is ED (erectile dysfunction), which has been a difficult experience in a new relationship. I cut back on my dosage. No adverse affects of increased anxiety. Better results with intimacy.
I take 1 mg in the afternoon. It makes me need a nap, but we bipolar peeps need naps anyway, so I don't feel so bad. But coming off of it is tough; you could actually die. So it looks like I'll be taking it forever... [Note: I have never read th...
Like most of you, I take aspirin daily, 162.5mg (it used to be 325 until my stomach rebelled). Most cardiologists recommend aspirin for heart disease sufferers.
Aspirin works by interfering with the generation of thromboxane A2 (TXA2) which is needed for platelet aggregation (clotting). The COX-1 enzyme acts on arachidonic acid (AA) to produce endoperoxides that in turn produce TXA2. Aspirin interferes with the generation of TXA2 by irreversably acetylating the platelet COX-1 enzyme thereby blocking its access to AA. Because platelets are anucleate , they cannot generate additional COX-1. In the absence of TXA2, platelet aggregation does not occur. Got all that?! Most practitioners prescribe anywhere from 81mg to 325mg for heart patients. Studies such as CURE suggest 81mg is optimal. The ISIS-2 study puts the dose at 162mg (for recent heart attack sufferers) and, frankly, since aspirin is so cheap, many simply make the leap to "more must be better." Ahh, but there are downsi...
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